Practical Geriatrics ›› 2023, Vol. 37 ›› Issue (12): 1270-1273.doi: 10.3969/j.issn.1003-9198.2023.12.019
Previous Articles Next Articles
Received:
2023-02-03
Online:
2023-12-20
Published:
2023-12-12
CLC Number:
[1] BAJPAI G, BREDEMEYER A, LI W, et al. Tissue resident CCR2- and CCR2+ cardiac macrophages differentially orchestrate monocyte recruitment and fate specification following myocardial injury[J] .Circ Res, 2019, 124(2):263-278. [2] HOFFMAN D, TEVET Y, TRZEBANSKI S, et al. A non-classical monocyte-derived macrophage subset provides a splenic replication niche for intracellular Salmonella[J]. Immunity, 2021, 54(12):2712-2723.e6. [3] HASHIMOTO D, CHOW A, NOIZAT C, et al. Tissue-resident macrophages self-maintain locally throughout adult life with minimal contribution from circulating monocytes[J]. Immunity, 2013, 38(4):792-804. [4] CHEN B, BRICKSHAWANA A, FRANGOGIANNIS N G. The functional heterogeneity of resident cardiac macrophages in myocardial injury[J]. Circ Res, 2019, 124(2):183-185. [5] JIA D L, CHEN S Q, BAI P Y, et al. Cardiac resident macrophage-derived legumain improves cardiac repair by promoting clearance and degradation of apoptotic cardiomyocytes after myocardial infarction[J]. Circulation, 2022, 145(20):1542-1556. [6] BAJPAI G, SCHNEIDER C, WONG N, et al. The human heart contains distinct macrophage subsets with divergent origins and functions[J]. Nat Med, 2018, 24(8):1234-1245. [7] MA Y, MOUTON A J, LINDSEY M L. Cardiac macrophage biology in the steady-state heart, the aging heart, and following myocardial infarction[J]. Transl Res, 2018, 191:15-28. [8] JIAN Y H, ZHOU X, SHAN W J, et al. Crosstalk between macrophages and cardiac cells after myocardial infarction[J]. Cell Commun Signal, 2023, 21(1):109. [9] HILGENDORF I, GERHARDT L M S, TAN T C, et al. Ly-6Chigh monocytes depend on Nr4a1 to balance both inflammatory and reparative phases in the infarcted myocardium[J]. Circ Res, 2014, 114(10):1611-1622. [10] JENKINS S J, RUCKERL D, COOK P C, et al. Local macrophage proliferation, rather than recruitment from the blood, is a signature of TH2 inflammation[J]. Science, 2011, 332(6035):1284-1288. [11] LEID J, CARRELHA J, BOUKARABILA H, et al. Primitive embryonic macrophages are required for coronary development and maturation[J]. Circ Res, 2016, 118(10):1498-1511. [12] WONG N R, MOHAN J, KOPECKY B J,et al. Resident cardiac macrophages mediate adaptive myocardial remodeling[J]. Immunity, 2021, 54(9):2072-2088.e7. [13] REVELO X S, PARTHIBAN P, CHEN C, et al. Cardiac resident macrophages prevent fibrosis and stimulate angiogenesis[J]. Circ Res, 2021, 129(12):1086-1101. [14] NICOLÁS-ÁVILA J A, LECHUGA-VIECO A V, ESTEBAN-MARTNEZ L,et al. A network of macrophages supports mitochondrial homeostasis in the heart[J]. Cell, 2020, 183(1):94-109.e23. [15] SUGITA J, FUJIU K, NAKAYAMA Y, et al. Crosstalk between cytotoxic CD8+ T cells and stressed cardiomyocytes triggers development of interstitial cardiac fibrosis in hypertensive mouse hearts[J]. Front Immunol, 2022, 13:1040233. [16] HULSMANS M, CLAUSS S, XIAO L, et al. Macrophages facilitate electrical conduction in the heart[J]. Cell, 2017, 169(3):510-522.e20. [17] MILLS C D, KINCAID K, ALT J M, et al. Pillars Article: M-1/M-2 Macrophages and the Th1/Th2 Paradigm.J. Immunol. 2000. 164:6166-6173[J]. J Immunol, 2017, 199(7):2194-2201. [18] HEO G S, KOPECKY B, SULTAN D, et al. Molecular imaging visualizes recruitment of inflammatory monocytes and macrophages to the injured heart[J]. Circ Res, 2019, 124(6):881-890. [19] LEUSCHNER F, DUTTA P, GORBATOV R, et al. Therapeutic siRNA silencing in inflammatory monocytes in mice[J]. Nat Biotechnol, 2011, 29(11):1005-1010. [20] WANG K Y, SUN X Q, SUN Y, et al. Transcriptional regulation of macrophages in heart failure[J]. Front Cardiovasc Med, 2023, 10:1148041. [21] HUANG C K, DAI D P, XIE H Y, et al. Lgr4 governs a pro-inflammatory program in macrophages to antagonize post-infarction cardiac repair[J]. Circ Res, 2020, 127(8):953-973. [22] MIA M M, CIBI D M, ABDUL GHANI S A B, et al. YAP/TAZ deficiency reprograms macrophage phenotype and improves infarct healing and cardiac function after myocardial infarction[J]. PLoS Biol, 2020, 18(12):e3000941. [23] WANG N X, WANG W W, WANG X Q, et al. Histone lactylation boosts reparative gene activation post-myocardial infarction[J]. Circ Res, 2022, 131(11):893-908. [24] LIU C F, WILSON TANG W H W. Epigenetics in cardiac hypertrophy andheartfailure[J]. JACC Basic Transl Sci, 2019, 4(8):976-993. |
Viewed | ||||||
Full text |
|
|||||
Abstract |
|
|||||
|