[1] LITTON E, LIM J. Iron metabolism: an emerging therapeutic target in critical illness[J].Crit Care, 2019, 23(1):81.
[2] ULLAH Z, ULLAH M I, HUSSAIN S,et al. Determination of serum trace elements (Zn, Cu, and Fe) in Pakistani Patients with rheumatoid arthritis[J].Biol Trace Elem Res,2017, 175(1):10-16.
[3] BO L, LIU Z C, ZHONG Y B, et al. Iron deficiency anemia’s effect on bone formation in zebrafish mutant[J].Biochem Biophys Res Commun,2016, 475(3):271-276.
[4] HU X R, CAI X R, MA R S, et al. Iron-load exacerbates the severity of atherosclerosis via inducing inflammation and enhancing the glycolysis in macrophages[J].J cell physiol,2019, 234(10):18792-18800.
[5] LI G F, PAN Y Z, SIROIS P,et al. Iron homeostasis in osteoporosis and its clinical implications[J].Osteoporos Int,2012, 23(10):2403-2408.
[6] 赵晋英,曾佳佳,李艳伟.肺的铁稳态机制与肺疾病时铁代谢的变化[J].中国生物化学与分子生物学报,2020,36(9):1002-1012.
[7] CHE J M, YANG J C, ZHAO B, et al. The effect of abnormal iron metabolism on osteoporosis[J].Biol Trace Elem Res,2020, 195(2):353-365.
[8] 李玉翠,冯玫.铁代谢异常与慢性病贫血[J].山西医药杂志,2005, 34(1):36-37.
[9] SIMS N A, MARTIN T J. Osteoclasts provide coupling signals to osteoblast lineage cells through multiple mechanisms[J]. Annu Rev Physiol, 2020, 82:507-529.
[10] 严伟, 王中汉, 刘贺. 骨重建失衡导致骨质疏松发生的作用机制及其药物治疗策略[J]. 中国组织工程研究, 2020, 24(30):4866-4874.
[11] 郭丽银, 王红祥. 铁过载的诊治[J]. 临床内科杂志, 2019, 36(10):662-664.
[12] 肖志坚. 铁过载诊断与治疗的中国专家共识[J]. 中华血液学杂志, 2011(8):572-574.
[13] GUO J C, REN R Y, SUN K, et al. PERK controls bone homeostasis through the regulation of osteoclast differentiation and function[J]. Cell Death Dis, 2020, 11(10):847.
[14] ZHU L, HUA F, DING W G, et al. The correlation between the Th17/Treg cell balance and bone health[J]. Immun Ageing, 2020, 17:30.
[15] UDAGAWA N, KOIDE M, NAKAMURA M, et al. Osteoclast differentiation by RANKL and OPG signaling pathways[J]. J Bone Miner Metab, 2021, 39(1):19-26.
[16] ROODMAN G D. Osteoclasts pump iron[J]. Cell Metab, 2009, 9(5):405-406.
[17] JIA P, XU Y J, ZHANG Z L, et al. Ferric ion could facilitate osteoclast differentiation and bone resorption through the production of reactive oxygen species[J]. J Orthop Res, 2012, 30(11):1843-1852.
[18] 王啸. “铁蓄积”影响骨代谢的临床观察及分子机制[D]. 苏州:苏州大学, 2018.
[19] ZARJOU A, JENEY V, AROSIO P, et al. Ferritin ferroxidase activity: a potent inhibitor of osteogenesis[J]. J Bone Miner Res, 2010, 25(1):164-172.
[20] BALOGH E, TOLNAI E, NAGY Jr B, et al. Iron overload inhibits osteogenic commitment and differentiation of mesenchymal stem cells via the induction of ferritin[J]. Biochim Biophys Acta, 2016, 1862(9):1640-1649.
[21] 程韶, 舒冰, 赵永见, 等. 氧化应激对骨重建的影响[J]. 中国骨质疏松杂志, 2019, 25(10):1478-1482.
[22] WANG F, LYU H H, ZHAO B, et al. Iron and leukemia: new insights for future treatments[J]. J Exp Clin Cancer Res, 2019, 38(1):406.
[23] 陈津晶, 易海振, 赖永榕, 等. 去铁胺、去铁酮治疗重型β地中海贫血铁过载患者的临床研究[J]. 国际输血及血液学杂志, 2020, 43(2):148-154.
[24] ELALFY M, WALI Y A, QARI M, et al. Deviating from safety guidelines during deferiprone therapy in clinical practice may not be associated with higher risk of agranulocytosis[J]. Pediatr Blood Cancer, 2014, 61(5):879-884.
[25] HAGHPANAH S, ZAREI T, ZAHEDI Z, et al. Compliance and satisfaction with deferasirox (Exjade) compared with deferoxamine in patients with transfusion-dependent beta-thalassemia[J]. Hematology, 2014, 19(4):187-191.
[26] DALE J C, BURRITT M F, ZINSMEISTER A R. Diurnal variation of serum iron, iron-binding capacity, transferrin saturation, and ferritin levels[J]. AmJ Clin Pathol, 2002, 117(5):802-808.
[27] TOXQUI L, VAQUERO M P. Chronic iron deficiency as an emerging risk factor for osteoporosis: a hypothesis[J]. Nutrients, 2015, 7(4):2324-2344.
[28] ZHENG J M, MAO X L, LING J Q, et al. Low serum levels of zinc, copper, and iron as risk factors for osteoporosis: a meta-analysis[J]. Biol Trace Elem Res, 2014, 160(1):15-23.
[29] 黄研生, 牛兴邦, 史少岩, 等. 贫血与骨质疏松相关性的研究进展[J]. 中国全科医学, 2017, 20(15):1911-1914.
[30] MALCZEWSKA-LENCZOWSKA J, SITKOWSKI D, SURAŁA O, et al. The association between iron and vitamin D status in female elite athletes[J]. Nutrients, 2018, 10(2):167.
[31] ZHAO G Y, ZHAO L P, HE Y F, et al. A comparison of the biological activities of human osteoblast hFOB1. 19 between iron excess and iron deficiency[J]. Biol Trace Elem Res, 2012, 150(1/3):487-495. |
