[1] ALPERT J S, THYGESEN K, ANTMAN E, et al. Myocardial infarction redefined--a consensus document of The Joint European Society of Cardiology/American College of Cardiology Committee for the redefinition of myocardial infarction [J]. J Am Coll Cardiol, 2000, 36(3): 959-969. [2] LOZANO R, NAGHAVI M, FOREMAN K, et al. Global and regional mortality from 235 causes of death for 20 age groups in 1990 and 2010: a systematic analysis for the Global Burden of Disease Study 2010 [J]. Lancet, 2012, 380(9859): 2095-2128. [3] 胡盛寿, 高润霖, 刘力生, 等. 《中国心血管病报告2018》概要 [J]. 中国循环杂志, 2019, 34(3): 209-220. [4] HARDING C, HEUSER J, STAHL P. Receptor-mediated endocytosis of transferrin and recycling of the transferrin receptor in rat reticulocytes [J]. J Cell Biol, 1983, 97(2): 329-339. [5] SIMONS M, RAPOSO G. Exosomes--vesicular carriers for intercellular communication [J]. Curr Opin Cell Biol, 2009, 21(4): 575-581. [6] COLOMBO M, RAPOSO G, THÉRY C. Biogenesis, secretion, and intercellular interactions of exosomes and other extracellular vesicles [J]. Annu Rev Cell Dev Biol, 2014, 30:255-289. [7] RAPOSO G, STOORVOGEL W. Extracellular vesicles: exosomes, microvesicles, and friends [J]. J Cell Biol, 2013, 200(4): 373-383. [8] HOSHINO D, KIRKBRIDE K C, COSTELLO K, et al. Exosome secretion is enhanced by invadopodia and drives invasive behavior [J]. Cell Rep, 2013, 5(5): 1159-1168. [9] SINHA S, HOSHINO D, HONG N H, et al. Cortactin promotes exosome secretion by controlling branched actin dynamics [J]. J Cell Biol, 2016, 214(2): 197-213. [10] HSU C, MOROHASHI Y, YOSHIMURA S-I, et al. Regulation of exosome secretion by Rab35 and its GTPase-activating proteins TBC1D10A-C [J]. J Cell Biol, 2010, 189(2): 223-232. [11] PFEFFER S R. Unsolved mysteries in membrane traffic [J]. Annu Rev Biochem, 2007, 76:629-645. [12] LUARTE A, BÁTIZ L F, WYNEKEN U, et al. Potential therapies by stem cell-derived exosomes in CNS diseases: Focusing on the Neurogenic Niche [J]. Stem Cells Int, 2016, 2016:5736059. [13] XIN H, LI Y, CHOPP M. Exosomes/miRNAs as mediating cell-based therapy of stroke [J]. Front Cell Neurosci, 2014, 8:377. [14] LAKKARAJU A, RODRIGUEZ-BOULAN E. Itinerant exosomes: emerging roles in cell and tissue polarity [J]. Trends Cell Biol, 2008, 18(5): 199-209. [15] SATO-KUWABARA Y, MELO S A, SOARES F A, et al. The fusion of two worlds: non-coding RNAs and extracellular vesicles--diagnostic and therapeutic implications (Review) [J]. Int J Oncol, 2015, 46(1): 17-27. [16] GUDURIC-FUCHS J, O’CONNOR A, CAMP B, et al. Selective extracellular vesicle-mediated export of an overlapping set of microRNAs from multiple cell types [J]. BMC Genomics, 2012, 13:357. [17] SKOG J, WÜRDINGER T, VAN RIJN S, et al. Glioblastoma microvesicles transport RNA and proteins that promote tumour growth and provide diagnostic biomarkers [J]. Nat Cell Biol, 2008, 10(12): 1470-1476. [18] FABBRI M, PAONE A, CALORE F, et al. MicroRNAs bind to Toll-like receptors to induce prometastatic inflammatory response [J]. Proc Natl Acad Sci U S A, 2012, 109(31): E2110-E2116. [19] MUNICH S, SOBO-VUJANOVIC A, BUCHSER W J, et al. Dendritic cell exosomes directly kill tumor cells and activate natural killer cells via TNF superfamily ligands [J]. Oncoimmunology, 2012, 1(7): 1074-1083. [20] MULCAHY L A, PINK R C, CARTER D R F. Routes and mechanisms of extracellular vesicle uptake [J]. J Extracell Vesicles, 2014, 3. doi: 10.3402/jev.v3.24641. [21] TIAN T, ZHU Y L, HU F H, et al. Dynamics of exosome internalization and trafficking [J]. J Cell Physiol, 2013, 228(7): 1487-1495. [22] BARILE L, GHERGHICEANU M, POPESCU L M, et al. Ultrastructural evidence of exosome secretion by progenitor cells in adult mouse myocardium and adult human cardiospheres [J]. J Biomed Biotechnol, 2012, 2012:354605. [23] MANOLE C G, CISMASIU V, GHERGHICEANU M, et al. Experimental acute myocardial infarction: telocytes involvement in neo-angiogenesis [J]. J Cell Mol Med, 2011, 15(11): 2284-2296. [24] KUWABARA Y, ONO K, HORIE T, et al. Increased MicroRNA-1 and MicroRNA-133a levels in serum of patients with cardiovascular disease indicate myocardial damage [J]. Circ Cardiovasc Genet, 2011, 4(4): 446-454. [25] LOYER X, ZLATANOVA I, DEVUE C, et al. Intra-cardiac release of extracellular vesicles shapes inflammation following myocardial infarction [J]. Circ Res, 2018, 123(1): 100-106. [26] WANG C, ZHANG C, LIU L, et al. Macrophage-derived mir-155-containing exosomes suppress fibroblast proliferation and promote fibroblast inflammation during cardiac injury [J]. Mol Ther, 2017, 25(1): 192-204. [27] BARILE L, LIONETTI V, CERVIO E, et al. Extracellular vesicles from human cardiac progenitor cells inhibit cardiomyocyte apoptosis and improve cardiac function after myocardial infarction [J]. Cardiovasc Res, 2014, 103(4): 530-541. [28] IBRAHIM A G, CHENG K, MARBÁN E. Exosomes as critical agents of cardiac regeneration triggered by cell therapy [J]. Stem Cell Reports, 2014, 2(5): 606-619. [29] ARSLAN F, LAI R C, SMEETS M B, et al. Mesenchymal stem cell-derived exosomes increase ATP levels, decrease oxidative stress and activate PI3K/Akt pathway to enhance myocardial viability and prevent adverse remodeling after myocardial ischemia/reperfusion injury [J]. Stem Cell Res, 2013, 10(3): 301-312. |