Practical Geriatrics ›› 2023, Vol. 37 ›› Issue (10): 989-994.doi: 10.3969/j.issn.1003-9198.2023.10.004
Previous Articles Next Articles
Received:2023-08-10
Online:2023-10-20
Published:2023-10-19
CLC Number:
| [1] FELSON D T, NAIMARK A, ANDERSON J, et al. The prevalence of knee osteoarthritis in the elderly. The Framingham Osteoarthritis Study[J]. Arthritis Rheum, 1987,30(8):914-918. [2] LIEBEN L. Osteoarthritis[J]. Nat Rev Dis Primers,2016,12(2):70-71. [3] HEINEGÅRD D, SAXNE T. The role of the cartilage matrix in osteoarthritis[J]. Nat Rev Rheumatol, 2011,7(1):50-56. [4] ANDRIACCHI T P, FAVRE J. The nature of in vivo mechanical signals that influence cartilage health and progression to knee osteoarthritis[J]. Curr Rheumatol Rep,2014,16(11):463. [5] LEE A S, ELLMAN M B, YAN D, et al. A current review of molecular mechanisms regarding osteoarthritis and pain[J]. Gene, 2013,527(2):440-447. [6] MCCULLOCH K, LITHERLAND G J, RAI T S, et al. Cellular senescence in osteoarthritis pathology[J]. Aging Cell, 2017,16(2):210-218. [7] CORYELL P R, DIEKMAN B O,LOESER R F, et al. Mechanisms and therapeutic implications of cellular senescence in osteoarthritis[J]. Nat Rev Rheumatol,2021,17(1):47-57. [8] CARAMES B, OLMER M, KIOSSES W B, et al. The relationship of autophagy defects to cartilage damage during joint aging in a mouse model[J]. Arthritis Rheumatol, 2015,67(6):1568-1576. [9] DALLAS S L, PRIDEAUX M,BONEWALD L F. The osteocyte: an endocrine cell... and more[J]. Endocr Rev, 2013, 34(5):658-690. [10] MURAKAMI T, ISHIDA T, TANAKA S. et al. Inflammation and subsequent nociceptor sensitization in the bone marrow are involved in an animal model of osteoarthritis pain[J]. Life Sci, 2023,324:121736. [11] SCANZELLO C R,GOLDRING S R. The role of synovitis in osteoarthritis pathogenesis[J]. Bone, 2012,51 (2):249-257. [12] KAPOOR M, MARTEL-PELLETIER J, LAJEUNESSE D, et al. Role of proinflammatory cytokines in the pathophysiology of osteoarthritis[J]. Nat Rev Rheumatol, 2011,7(1):33-42. [13] BASBAUM A I, BAUTISTA D M, SCHERRER G,et al. Celluar and molecular mechanisms of pain[J]. Cell, 2009, 139(2): 267-284. [14] OBEIDAT A M,WOOD M J,ADAMCZYD N S,et al. Piezo2 expressing nociceptors mediate mechanical sensitization in experimental osteoarthritis[J]. Nat Commun, 2023,14(1):2479. [15] CAMPBELL J N,MEYER R A. Mechanisms of neuropathic pain[J]. Neuron, 2006,52(1):77-92. [16] DAGNINO A P A,CAMPOS M M. Chronic pain in the elderly: mechanisms and perspectives[J]. Front Hum Neurosci, 2022,16:736688. [17] FU K, ROBBINS S R,MCDOUGALL J J. Osteoarthritis: the genesis of pain[J]. Rheumatology:Oxford, 2018,57(Suppl 4):iv43-iv50. [18] NEOGI T, NEVITT MC, YANG M,et al. Consistency of knee pain: correlates and association with function[J]. Osteoarthritis Cartilage,2010,18(10):1250-1255. [19] WOOD M J, MILLER R E,MALFAIT A M. The genesis of pain in osteoarthritis: inflammation as a mediator of osteoarthritis pain[J]. Clin Geriatr Med, 2022, 38(2):221-238. [20] YEATER T D, CRUZ C J, CRUZ-ALMEIDA Y,et al. Autonomic nervous system dysregulation and osteoarthritis pain: mechanisms, measurement, and future outlook[J]. Curr Rheumatol Rep, 2022, 24(6):175-183. [21] ZHU S, ZHU J, ZHEN G, et al. Subchondral bone osteoclasts induce sensory innervation and osteoarthritis pain[J]. J Clin Invest, 2019,129(3):1076-1093. [22] ASHRAF S,WIBBERLEY H,MAPP P L,et al. Increased vascular penetration and nerve growth in the meniscus: a potential source of pain in osteoarthritis[J]. Ann Rheum Dis, 2011,70(3):523-529. [23] WALSH D A,MCWILLIAM D F,TURLEY M J,et al. Angiogenesis and nerve growth factor at the osteochondral junction in rheumatoid arthritis and osteoarthritis[J]. Rheumatology:Oxford, 2010,49(10):1852-1861. [24] OHASHI Y, UCHIDA K, FUKUSHIMA K,et al. Mechanisms of peripheral and central sensitization in osteoarthritis pain[J]. Cureus, 2023,15(2):e35331. [25] MIOTLA ZAREBSKA J,CHANALARIS A DRISCOLL C,et al. CCL2 and CCR2 regulate pain-related behaviour and early gene expression in post-traumatic murine osteoarthritis but contribute little to chondropathy[J]. Osteoarthritis Cartilage, 2017,25(3):406-412. [26] ISHIHARA S,OBEIDAT A M,WOKOSIN D, et al. The role of intra-articular neuronal CCR2 receptors in knee joint pain associated with experimental osteoarthritis in mice[J]. Arthritis Res Ther, 2021, 23(1):103. [27] WOODELL-MAY J E,SOMMERFELD S D. Role of inflammation and the immune system in the progression of osteoarthritis[J]. J Orthop Res, 2020,38(2):253-257. [28] OHASHI Y,UCHIDA K, FUKUSHIMA K,et al. NGF expression and elevation in hip osteoarthritis patients with pain and central sensitization[J]. Biomed Res Int, 2021:9212585. [29] Bonnington J K,McNaughton P A. Signalling pathways involved in the sensitisation of mouse nociceptive neurones by nerve growth factor[J]. J Physiol, 2003,551(Pt 2):433-446. [30] ZHANG Y H, VASKO M R,NICOL G D. Ceramide, a putative second messenger for nerve growth factor, modulates the TTX-resistant Na(+) current and delayed rectifier K(+) current in rat sensory neurons[J]. J Physiol, 2002,544(2):385-402. [31] SUN Q, ZHEN G, LI T P, et al. Inhibition of PGE2 in subchondral bone attenuates osteoarthritis[J]. Cells, 2022,11(17):2760. [32] ENGLAND S, BEVAN S,DOCHERTY R J. PGE2 modulates the tetrodotoxin-resistant sodium current in neonatal rat dorsal root ganglion neurones via the cyclic AMP-protein kinase A cascade[J]. J Physiol, 1996,495(Pt 2):429-440. [33] Miller R E, TRAN P B, DAS R, et al. CCR2 chemokine receptor signaling mediates pain in experimental osteoarthritis[J]. Proc Natl Acad Sci U S A, 2012,109(50):20602-20607. [34] RICHTER F,NATURA G,GOSER S,et al. Tumor necrosis factor causes persistent sensitization of joint nociceptors to mechanical stimuli in rats[J]. Arthritis Rheum, 2010,62(12):3806-3814. [35] RAOOF R, MARTIN GIL C, LAFEBER F P J G, et al. Dorsal root ganglia macrophages maintain osteoarthritis pain[J]. J Neurosci, 2021,41(39): 8249-8261. [36] YU X,, LIU H, HAMEL K A, et al. Dorsal root ganglion macrophages contribute to both the initiation and persistence of neuropathic pain[J]. Nat Commun, 2020,11(1):264. [37] MEASE P J, HANNA S, FRAKES E P,et al. Pain mechanisms in osteoarthritis: understanding the role of central pain and current approaches to its treatment[J]. J Rheumatol, 2011,38(8):1546-1551. [38] PAN T T, PAN F, GAO W, et al. Involvement of macrophages and spinal microglia in osteoarthritis pain[J]. Curr Rheumatol Rep, 2021,23(5):29. [39] ZIEGLGANSBERGER W. Substance P and pain chronicity[J]. Cell Tissue Res, 2019,375(1):227-241. [40] IYENGAR S, OSSIPOV M H, JOHNSON K W. The role of calcitonin gene-related peptide in peripheral and central pain mechanisms including migraine[J]. Pain, 2017,158(4):543-559. [41] BJURSTROM M F,BLENNOW K,ZETTERBERG H,et al. Central nervous system monoaminergic activity in hip osteoarthritis patients with disabling pain: associations with pain severity and central sensitization[J]. Pain Rep, 2022,7(1):e988. [42] FUJII T, MASHIMO M, MORIWAKI Y, et al. Expression and function of the cholinergic system in immune cells[J]. Front Immunol, 2017,8:1085. [43] GIBSON S J, FARRELL M. A review of age differences in the neurophysiology of nociception and the perceptual experience of pain[J]. Clin J Pain, 2004, 20(4):227-239. [44] EL TUMI H, JOHNSON M I, DANTAS P B F, et al. Age-related changes in pain sensitivity in healthy humans: a systematic review with meta-analysis[J]. Eur J Pain, 2017,21(6):955-964. [45] WEYER A D, ZAPPIA K J, GARRISON S R,et al. Nociceptor sensitization depends on age and pain chronicity(1,2,3) [J]. eNeuro, 2016, 3 (1):115. [46] TINNIRELLO A, MAZZOLENI S,SANTI C. Chronic pain in the elderly: mechanisms and distinctive features[J]. Biomolecules, 2021,1(8):1256. [47] FULLERTON E F, RUBAHARAN M, KAROM M C, et al. Advanced age attenuates the antihyperalgesic effect of morphine and decreases mu-opioid receptor expression and binding in the rat midbrain periaqueductal gray in male and female rats[J]. Neurobiol Aging, 2021, 98:78-87. [48] DA SILVA J T, TRICOU C, ZHANG Y, et al. Brain networks and endogenous pain inhibition are modulated by age and sex in healthy rats[J]. Pain, 2020,161(6):1371-1380. [49] RO J Y,ZHANG Y P,TRICOU C,et al. Age and sex differences in acute and osteoarthritis-like pain responses in rats[J]. J Gerontol A Biol Sci Med Sci, 2020,75(8):1465-1472. [50] PETERSEN K K, ARENDT-NIELSEN L, FINOCCHIETTI S, et al. Age interactions on pain sensitization in patients with severe knee osteoarthritis and controls[J]. Clin J Pain, 2017,33(12):1081-1087. [51] TAVARES D R B, MOÇA TREVISANI V F, FRAZAO OKAZAKI J E, et al. Risk factors of pain, physical function, and health-related quality of life in elderly people with knee osteoarthritis: a cross-sectional study[J]. Heliyon, 2020,6(12):e05723. [52] GONZALEZ-ROLDAN A M, TERRASA J L, SITGES C, et al. Alterations in neural responses and pain perception in older adults during distraction[J]. Psychosom Med,2020, 82(9):869-876. |
| No related articles found! |
| Viewed | ||||||
|
Full text |
|
|||||
|
Abstract |
|
|||||
|
